NCLEX CRUSADE ACADEMY TEST - 15 EKG PRT 2

1. First-degree AV block is characterized by: Dropped QRS complexes. Progressive PR interval prolongation. PR interval greater than 0.20 seconds but constant. Complete atrial and ventricular dissociation.

2. Second-degree AV block Type I (Wenckebach) is identified by: Constant PR interval with dropped QRS. Progressive PR interval prolongation followed by a dropped QRS. Irregular P waves. Absence of QRS complexes.

3. Second-degree AV block Type II (Mobitz II) is characterized by: Random dropped QRS complexes without PR prolongation. Constant PR interval with occasional dropped QRS. Absence of P waves. Short PR interval.

4. Third-degree AV block (complete heart block) occurs when: PR interval shortens progressively. P waves disappear. Atria and ventricles beat independently. Only the ventricles contract.

5. The first-line medication for symptomatic bradycardia is: Adenosine. Amiodarone. Atropine. Lidocaine.

6. If atropine fails in severe bradycardia, the next intervention is often: Adenosine. Transcutaneous pacing. Defibrillation. Magnesium sulfate.

7. Atrial fibrillation is characterized by: Regular rhythm. Sawtooth pattern. Irregularly irregular rhythm. Wide QRS complexes.

8. Atrial flutter is identified by: Irregular baseline. Sawtooth flutter waves. Wide QRS complexes. Absence of P waves.

9. A major complication of atrial fibrillation is: Pulmonary edema. Stroke due to clot formation. Bradycardia. Hypokalemia.

10. Medications commonly used for rate control in atrial fibrillation include: Calcium channel blockers and beta blockers. Antibiotics. Insulin. Corticosteroids.

11. The drug of choice for SVT is: Dopamine. Adenosine. Lidocaine. Digoxin.

12. Adenosine administration typically results in: Permanent cardiac arrest. Brief asystole followed by rhythm reset. Severe tachycardia. Hypertension.

13. PVCs (Premature Ventricular Contractions) originate from: SA node. Atria. Ventricles. AV node.

14. Multifocal PVCs are considered more dangerous because: They originate from multiple ventricular sites. They occur only during sleep. They occur in atria. They always produce bradycardia.

15. The R-on-T phenomenon occurs when: A PVC occurs during ventricular repolarization. A P wave overlaps a QRS. The T wave disappears. Two QRS complexes merge.

16. Ventricular tachycardia is defined as: Rapid atrial rhythm. Rapid ventricular rhythm originating in ventricles. Slow ventricular rhythm. Irregular atrial rhythm.

17. If a patient with ventricular tachycardia has no pulse, the correct intervention is: Synchronized cardioversion. Defibrillation and CPR. Adenosine. Vagal maneuvers.

18. Torsades de Pointes is commonly treated with: Atropine. Magnesium sulfate. Adenosine. Dopamine.

19. Ventricular fibrillation results in: Low cardiac output. Zero cardiac output. Increased cardiac output. Mild hypotension.

20. The priority treatment for ventricular fibrillation is: Cardioversion. Defibrillation. Adenosine. Atropine.

21. Synchronized cardioversion differs from defibrillation because: It delivers a lower shock. It synchronizes with the R wave. It is used only during cardiac arrest. It requires CPR.

22. Defibrillation is used for: Atrial fibrillation. Stable tachycardia. Pulseless VT and ventricular fibrillation. Bradycardia.

23. After cardiac catheterization via femoral artery, the patient should: Sit upright. Walk immediately. Lie supine with leg straight. Flex the hip.

24. A key NCLEX prioritization rule when interpreting EKG is: Always treat the rhythm first. Treat the patient, not the monitor. Ignore symptoms. Always shock abnormal rhythms.

25. Before treating any rhythm abnormality the nurse must: Call the physician. Check for a pulse. Start IV fluids. Obtain consent.